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Patients With Rheumatoid Arthritis Who Had Poor Response To Other Drugs Could Have Better Results With Golimumab
An article published Online First and in this week"s edition of reports information about Golimumab, a new tumour necrosis factor-í± (TNF-í±) inhibitor. It reduces the signs and symptoms of rheumatoid arthritis in patients who have previously received any other TNF-í± inhibitor. This drug might be a good alternative for patients who have inadequate responses to one or two other TNF-í± inhibitors.
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InVitria To Unveil Powerful Cell Culture Media Component ZAP-CHO
InVitria"s Director of Cell Culture, Dr. Steve Pettit, will present the performance enhancing features of ZAP-CHO, a new media component for Chinese hamster ovary (CHO) cell culture, at 3:30 pm on Wednesday, May 20, 2009 as part of BioProcess International"s BioProcess Theatre, a venue employed to facilitate the sharing of advances and discoveries in the biopharmaceutical sector.
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Pitt Team Reports In Nature That Unique Portion Of Enzyme Fights Lung Infection
An enzyme known to play a key role in the development of emphysema serves as the first line of defense against bacterial infection of the lung, according to researchers at the University of Pittsburgh School of Medicine. They also found that the antimicrobial activity comes from a small portion of the enzyme that is structurally and sequentially unique in nature.
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Comment On Dimebolin (dimebon) Increasing Brain Beta Amyloid In Mouse Models

Dimebolin, originally administered as an antihistamine, has previously been found to help slow the decline of cognitive ability in participants. This study analysed tau and how dimebolin affects build up of the toxic beta amyloid protein, which is widely believed to be linked to the development of dementia. This research showed that while dimebolin had a beneficial effect it actually caused an increase of the beta amyloid in mouse models. "This head scratching research has thrown up surprising results and suggests that while dimebolin provides cognitive benefits it may actually increase levels of toxic beta amyloid in the brain. One possible explanation may be that dimebolin is accelerating the process of beta amyloid development, which may reduce its toxicity. The questions surrounding whether beta amyloid causes nerve death in Alzheimer"s or is a bi-product of the process must be addressed by further research. "One in three people over 65 will die with dementia. Further, more robust research is needed to clarify whether dimebolin is an effective treatment for Alzheimer"s in the long term and if these puzzling increases in beta amyloid are also seen in human brains. With the right investment, dementia can be defeated. Alzheimer"s Society funds research to improve the lives of people today and to find a cure for tomorrow." Professor Clive Ballard Director of Research Alzheimer"s Society Reference: Samuel Gandy, Dimebon regulates amyloid beta metabolism in cultured cells, isolated nerve terminals and interstitial fluid of living rodent brain. Alzheimer"s Society


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