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Identifying Genetic Markers Related To Drug Induced Liver Injury
The International Serious Adverse Events Consortium (SAEC) has announced initial results from its research designed to discover genetic markers that may predict individuals at risk for serious drug induced liver injury (DILI). The SAEC is a nonprofit research corporation, launched in the fall of 2007, comprised of and funded by 10 leading pharmaceutical companies and the Wellcome Trust. The U.S. Food and Drug Administration (FDA) also contributes to the scientific and strategic direction of this novel research effort. The collection and initial characterization of the DILI cases supporting these results was performed by the UK-based DILIGEN network, led by Professor Ann Daly and colleagues at Newcastle University, but also involving researchers at the University of Liverpool and at Queen"s Medical Centre, Nottingham.
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Scientists Closer To Making Implantable Bone Material, Thanks To New Research
Scientists are closer to understanding how to grow replacement bones with stem cell technology, thanks to research published in the journal Nature Materials.
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The Right Messenger For A Healthy Immune Response
Researchers from the Molecular Immunology group at the Helmholtz Centre for Infection Research (HZI) in Braunschweig, Germany have now shown that Beta-Interferon also plays a crucial role during an immune response: without Beta-Interferon immune cells are unable to show "wanted posters" of pathogens to other cells. As a consequence, these cells will not recognize the pathogen and the immune response does not start properly. The group"s results have now been published in the current issue of the scientific magazine Journal of Immunology.
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Deaths From Heart Disease In Canada Decreased 30 Percent: 10-year National Study

Rates of death and hospital admissions for cardiovascular disease declined 30% over a 10-year period in Canada, according to a new study in CMAJ (Canadian Medical Association Journal), pointing to successful efforts to prevent heart disease, the leading cause of death globally. However, for the first time, more women than men are dying of cardiovascular causes. The study, the first of its kind in Canada, looked at data from the Canadian Mortality Database, Statistics Canada"s national death registry which contains information on the cause of all deaths in the country. It also looked at hospital admissions for heart attacks, heart failure and stroke. A major finding was the rapid decline in death rates from heart attacks, with 4000 fewer Canadians dying from acute myocardial infarction in 2004 than in 1994. This could reflect declines in risk factors such as smoking and increased use of statins to control cholesterol. However, the study showed high rates of death and hospital admission related to cardiovascular disease in elderly women. "This highlights the need for increased investment in education and research on cardiovascular health and disease in women," write Dr. Jack Tu from the Institute for Clinical Evaluative Sciences (ICES) and coauthors. The authors caution that despite the 30% decrease, "these findings are not grounds for complacency. They suggest that previous efforts to prevent cardiovascular events have been successful, but in many cases they may have delayed the occurrence of such events until people are older and potentially more difficult to treat." In a related commentary, Dr. Simon Capewell and Dr. Martin O"Flaherty from the University of Liverpool, United Kingdom, write that global reductions in cardiovascular disease are due to success in reducing risk factors as well as treatment of heart disease. They caution that patients with cardiovascular disease in the future will be older and more challenging to treat. "Prevention, therefore, becomes vital because over 80% of premature cardiovascular disease is avoidable," state the authors. Promotion of smoking cessation, healthier diets and physical activity is crucial in addition to medications that control blood pressure and cholesterol. Kim Barnhardt Canadian Medical Association Journal


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